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Correction for Karenko et al., Cancer Res 65 (18) 8101-8110.
Cancer Research 68, 7692, September 15, 2008. doi: 10.1158/0008-5472.CAN-68-18-COR1
© 2008 American Association for Cancer Research
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Corrections

Correction: NAV3 Gene Deletion/Translocation in CTCL

In the article on NAV3 gene deletion/translocation in CTCL in the September 15, 2005 issue of Cancer Research (1), inactivation of the NAV3/POMFIL1 gene on chromosome 12 by deletion or translocation was found to be associated with and suggested to be causative for cutaneous T-cell lymphoma. A putative tumor suppressive role of NAV3/POMFIL1 was formerly also suggested by Coy et al. in 2002 (2). Coy et al. described a translocation event affecting the chromosomal regions 1p and 12q which resulted in an inactivation of NAV3/POMFIL1 (2).

References

  1. Karenko L, Hahtola S, Päivinen S, Karhu R, Syrjä S, Kähkönen M, Nedoszytko B, Kytölä S, Zhou Y, Blazevic V, Pesonen M, Nevala H, Nupponen N, Sihto H, Krebs I, Poustka A, Roszkiewicz J, Saksela K, Peterson P, Visakorpi T, Ranki A. Primary cutaneous T-cell lymphomas show a deletion or translocation affecting NAV3, the human UNC-53 homologue. Cancer Res 2005;65:8101–10.[Abstract/Free Full Text]
  2. Coy JF, Wiemann S, Bechmann I, Bachner D, Nitsch R, Kretz O, Christiansen H, Poustka A. Pore membrane and/or filament interacting like protein 1 (POMFIL1) is predominantly expressed in the nervous system and encodes different protein isoforms. Gene 2002;290:73–94.[CrossRef][Medline]




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HOME HELP FEEDBACK SUBSCRIPTIONS ARCHIVE SEARCH TABLE OF CONTENTS
Cancer Research Clinical Cancer Research
Cancer Epidemiology Biomarkers & Prevention Molecular Cancer Therapeutics
Molecular Cancer Research Cancer Prevention Research
Cancer Prevention Journals Portal Cancer Reviews Online
Annual Meeting Education Book Meeting Abstracts Online