More articles from Poster Presentations - Enhancement of Apoptosis by Chemotherapeutic Agents

Abstract 230: LMP1 function in NK/T-cell lymphoma
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Cancer Research Apr 2010, 70 (8 Supplement) 230; DOI: 10.1158/1538-7445.AM10-230
Abstract 210: Glionitrin A causes apoptosis and DNA damage-induced S phase arrest in human prostate cancer cells
Young-Joo Kim, Hak Cheol Kwon, Ji-Hye Yoo, Hyun Ok Yang

Cancer Research Apr 2010, 70 (8 Supplement) 210; DOI: 10.1158/1538-7445.AM10-210
Abstract 208: Low-dose actinomycin D decreases Mcl-1 expression and acts synergistically with ABT-737 to induce apoptosis of SCLC cell lines
Haishan Xu, Malika Humphries, Geoffrey W. Krystal

Cancer Research Apr 2010, 70 (8 Supplement) 208; DOI: 10.1158/1538-7445.AM10-208
Abstract 213: Combinative treatment for prostate cancer by adenoviral-mediated gene therapy expressing a novel apoptosis gene and chemotherapy
Yi Lu, Ben Beheshti, Xiongwen Zhang

Cancer Research Apr 2010, 70 (8 Supplement) 213; DOI: 10.1158/1538-7445.AM10-213
Abstract 227: Antitumor activity of glyceollins through induction of apoptosis
Hyo Jung Kim, Jiyeon Seo, Ji Sun Lim, Jia Park, Dae Hwan Nam, Annalene Garcia, Jong-Sang Kim

Cancer Research Apr 2010, 70 (8 Supplement) 227; DOI: 10.1158/1538-7445.AM10-227
Abstract 223: Apoptotic activity of Polygonum cuspidatum in KB human oral cancer cells through the downregulation of specificity protein 1
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Cancer Research Apr 2010, 70 (8 Supplement) 223; DOI: 10.1158/1538-7445.AM10-223
Abstract 224: Antitumorigenic effect of synthetic C-substituted diindolyl methanes originating from cruciferous vegetables in KB human oral cancer cells through endoplasmic reticulum stress
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Cancer Research Apr 2010, 70 (8 Supplement) 224; DOI: 10.1158/1538-7445.AM10-224
Abstract 222: Inhibition of cellular FLICE-inhibitory protein and induction of death receptor 5 expression is a major component of δ-tocotrienol-induced apoptosis in pancreatic cancer cells
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Abstract 211: New synthetic flavone exerts antitumor activity on leukemia cell lines
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Abstract 217: Vitamin E forms induce apoptosis, necrosis, and autophagy and caused accumulation of sphingolipid intermediates in human colon cancer cells
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