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Volume 80, Issue 17, pp. 3606-3619
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History
- Received January 13, 2020
- Revision received May 9, 2020
- Accepted June 29, 2020
- Published first July 8, 2020.
Article Versions
- Previous version (July 8, 2020 - 07:41).
- You are viewing the most recent version of this article.
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©2020 American Association for Cancer Research.
Author Information
- Fatouma Alimirah1,
- Tanya Pulido1,
- Alexis Valdovinos1,
- Sena Alptekin1,2,
- Emily Chang1,
- Elijah Jones1,
- Diego A. Diaz1,
- Jose Flores1,
- Michael C. Velarde1,3,
- Marco Demaria1,4,
- Albert R. Davalos1,
- Christopher D. Wiley1,
- Chandani Limbad1,
- Pierre-Yves Desprez1, and
- Judith Campisi1,5,*
- 1Buck Institute for Research on Aging, Novato, California.
- 2Dokuz Eylul University School of Medicine, Izmir, Turkey.
- 3Institute of Biology, University of the Philippines Diliman, College of Science, Quezon City, Philippines.
- 4European Research Institute for the Biology of Ageing, University Medical Center Groningen, Groningen, the Netherlands.
- 5Biosciences Division, Lawrence Berkeley National Laboratory, Berkeley, California.
- ↵*Corresponding Author:
Judith Campisi, Buck Institute for Research on Aging, 8001 Redwood Boulevard, Novato, CA 94945. Phone: 415-209-2066; E-mail: jcampisi{at}buckinstitute.org
September 2020
Volume 80, Issue 17
Volume 80, Issue 17
Cellular Senescence Promotes Skin Carcinogenesis through p38MAPK and p44/42MAPK Signaling
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